https://www.selleckchem.com/products/ipi-549.html
MiR-26a-5p promoted LC3II and decreased p62 expression in PE-induced cardiac hypertrophy in the presence or absence of lysosomal inhibitor. Furthermore, miR-26a-5p significantly inhibited GSK3 expression in vitro and in vivo. Dual luciferase report results confirmed that miR-26a-5p could directly target GSK3 . GSK3 overexpression significantly reversed the expression of cardiac hypertrophy-related markers including ANP, ACTA1 and MYH7. Immunofluorescence staining results demonstrated that miR-26a-5p promoted cardiac hypertrophy related
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