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Our outcomes suggested that polydatin considerably inhibited proliferation of MG-63 cells in a dose- and time-dependent way, and enhanced their particular apoptosis and autophagic flux. Further experiments showed that polydatin decreased the phrase and phosphorylation (Y705) standard of STAT3 (Signal transducer and activator of transcription 3), increased the appearance of autophagy-related genetics (Atg12, Atg14, BECN1, PIC3K3), and therewith triggered autophagic cell death
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